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About the Cornerstone Relapse Prevention Model

Relapse Prevention Model
Source Info - PDF File (http://pubs.niaaa.nih.gov/publications/arh23-2/151-160.pdf)

Relapse, or the return to heavy Alcohol use following a period of abstinence or moderate use, occurs in many drinkers who have undergone alcoholism treatment. Traditional alcoholism treatment approaches often conceptualize relapse as an end-state, a negative outcome equivalent to treatment failure.

Thus, this perspective considers only a dichotomous treatment outcome—that is, a person is either abstinent or relapsed. In contrast, several models of relapse that are based on social-cognitive or behavioral theories emphasize relapse as a transitional process, a series of events that unfold over time (Annis 1986; Litman et al. 1979; Marlatt and Gordon 1985).

According to these models, the relapse process begins prior to the first posttreatment alcohol use and continues after the initial use. This conceptualization provides a broader conceptual framework for intervening in the relapse process to prevent or reduce relapse episodes and thereby improve treatment outcome.

This article presents one influential model of the antecedents of relapse and the treatment measures that can be taken to prevent or limit relapse after treatment completion. This relapse prevention (RP) model, which was developed by Marlatt and Gordon (1985) and which has been widely used in recent years, has been the focus of considerable research.

This article reviews variousimmediate and covert triggers of relapse proposed by the RP model, as well as numerous specific and general intervention strategies that may help patients avoid and cope with relapse-inducing situations. The article also presents studies that have provided support for the validity of the RP model. Overview of the RP Model Marlatt and Gordon’s (1985) RP model is based on social-cognitive psychology and incorporates both a conceptual model of relapse and a set of cognitive and behavioral strategies to prevent or limit relapse episodes (for a detailed description of the development, theoretical underpinnings, and treatment components of the RP model, see Dimeff and Marlatt 1998; Marlatt 1996; Marlatt and Gordon 1985). A central aspect of the model is the detailed classification (i.e., taxonomy) of factors or situations that can precipitate or con- tribute to relapse episodes.

In general, the RP model posits that those factors fall into two categories: immediate determinants (e.g., high-risk situations, a person’s coping skills, outcome expectancies, and the abstinence violation effect) and covert antecedents (e.g., lifestyle imbalances and urges and cravings). Treatment approaches based on the RP model begin with an assessment of the environmental and emotional char- acteristics of situations that are potentially associated with relapse (i.e., high-risk situations).

After identifying those characteristics, the therapist works forward by analyzing the individual drinker’s response to these situations, as well as backward to examine the lifestyle factors that increase the drinker’s exposure to high-risk situations. Based on this careful examination of the relapse process, the therapist then devises strategies to target weaknesses in the client’s cognitive and behavioral repertoire and thereby reduce the risk of relapse.

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